J Veterinary Medical Science 2009, 71:255–261.CrossRef 32. Mateo E, Cárcamo J, Urquijo M, Perales I, Fernández-Astorga A: Evaluation

of a PCR assay for the detection and identification of Campylobacter jejuni and Campylobacter coli in retail poultry products. Res Microbiol 2005, Proteasome inhibitor 156:568–574.PubMedCrossRef 33. Hochel I, Viochna D, Skvor J, Musil M: Development of an indirect competitive ELISA for detection of Campylobacter jejuni subsp. jejuni O:23 in foods. Folia Microbiol (Praha) 2004, 49:579–586.CrossRef 34. Ledergerber U, Regula G, Stephan R, Danuser J, Bissig B, Stärk KD: Risk factors for antibiotic resistance in Campylobacter spp. isolated from raw poultry meat in Switzerland. BMC Public Health 2003, 93:39.CrossRef 35. Oyarzabal OA, Liu L: Significance of sample weight and enrichment ratio on the isolation of Campylobacter spp. from retail broiler meat. J Food Prot 2010, 73:1339–1343.PubMed 36. Atanassova V, Ring C: Prevalence of Campylobacter spp. in poultry and poultry meat in Germany.

Int J Food Microbiol 1999, 51:187–190.PubMedCrossRef 37. Gurtler M, Alter T, Kasimir S, Fehlhaber K: The importance of Campylobacter coli in human campylobacteriosis: prevalence and genetic characterization. Epidemiol ITF2357 supplier Infect 2005, 133:1081–1087.PubMedCrossRef 38. Boysen L, Vigre H, Rosenquist H: Seasonal influence on the prevalence of thermotolerant Campylobacter in retail broiler meat in Denmark. Food Microbiol 2011, 28:1028–1032.PubMedCrossRef 39. Steinbrueckner B, Ruberg F, Kistv M: Bacterial much genetic fingerprint: a reliable factor in the study of the epidemiology of human Campylobacter enteritis? J Clin Microbiol 2001, 39:4155–4159.PubMedCrossRef

Competing interests The authors declare that no competing interests exist. Authors’ contributions AW collected and analyzed part of the samples and identified the isolates. AW performed the PFGE analysis. OAO conceived and coordinated the study and designed and revised the manuscript. All authors read and accepted the final version of the manuscript.”
“Background Entamoeba histolytica, a micro-aerophilic intestinal protozoan parasite and the causative agent of invasive amoebiasis (colitis and amoebic liver abscess), remains a significant cause of morbidity and mortality in developing countries [1]. It is well known that the parasite is constantly interacting with the intestinal gut flora however the contribution of the flora in the manifestation of the disease is poorly understood. The human gastrointestinal (GI) tract is VX-689 ic50 nutrient-rich environment packed with a complex and dynamic consortia of trillions of microbes [2].The vast majority reside in our colon where densities approach 1011 – 1012 cells/ml, the highest density recorded for any microbial habitat [3]. About 500–1000 bacterial species colonize the adult intestine,with 30–40 species comprising up to 97% of the total population [4, 5].